Citation

Zakhari R. J. Nurse Pract. 2014; 10(8): 616-619.

Copyright

(Copyright © 2014, Elsevier Publishing)

DOI

10.1016/j.nurpra.2014.07.001

PMID

unavailable

Abstract

Ethylene glycol poisoning is a major contributor to the development of idiopathic metabolic acidosis, which may lead to renal failure. Ethylene glycol poisoning should be among suspected differentials when assessing a seemingly intoxicated patient with hypocalcemia, anion gap acidosis, and nontoxic blood alcohol levels. Glycol intoxication may lead to delayed neurologic manifestations, which may lead to an inaccurate diagnosis. No clear clinical guidelines exist to recommend treatment for this late effect. This is a case of the complete resolution of facial nerve deficit secondary to glycol poisoning after the completion of a steroid therapy course on an inpatient psychiatric unit. © 2014 Elsevier Inc.

Language: en

Keywords

Suicide; Poisoning; Ethylene glycol; Toxicity; Metabolic acidosis; Ingestion; Fomepizole; Calcium oxalate crystals; Cranial nerve deficit; Neurological sequelae of glycol intoxication; Palsy