Citation

Koch HJ. Arch. Psychiatry Psychother. 2013; 15(2): 37-43.

Copyright

(Copyright © 2013, Polish Psychiatric Association)

DOI

10.12740/APP/18442

PMID

unavailable

Abstract

Psychiatric disease, particularly depression and stress disorders, worsen the outcome of cardiovascular disease substantially. Although this mind-heart interaction is known since the 1930s, many questions with regard to the underlying pathophysiology remain to be answered. Apart from psychological stress and psychiatric disease, inflammatory or psychoimmunology processes, metabolic or endocrinological mechanisms may be involved as are lifestyle and effects of drug treatment. The takotsubo or broken heart cardiomyopathy, which can be regularly referred to stressful event, may serve as paradigm to understand pathological base of the mind-heart relation.

Language: en

Keywords

human; Stress; Depression; suicide; dementia; depression; lifestyle; heart failure; C reactive protein; inflammation; obesity; article; mental disease; disease association; triacylglycerol; amfebutamone; citalopram; fluoxetine; mirtazapine; moclobemide; paroxetine; sertraline; quetiapine; panic; psychophysiology; coping behavior; acetylsalicylic acid; tranylcypromine; mental stress; cardiomyopathy; olanzapine; risperidone; amiodarone; cardiovascular disease; muscle hypertonia; hypercortisolism; amisulpride; heart arrhythmia; heart disease; hyperlipidemia; QT prolongation; low birth weight; warfarin; serotonin transporter; nitrate; sotalol; tyramine; dipeptidyl carboxypeptidase inhibitor; interleukin 6; omega 3 fatty acid; tumor necrosis factor alpha; aripiprazole; cardiovascular risk; heparin; insulin resistance; metabolic syndrome X; reactive oxygen metabolite; heart left ventricle failure; asenapine; takotsubo cardiomyopathy; Cardiomyopathy; 5,10 methylenetetrahydrofolate reductase (FADH2); Broken heart; Coping pathophysiology; hyperhomocysteinemia; Mind-heart interaction; syndrome X; Tako-tsubo; thrombocyte function