Citation

Rikani AA, Choudhry Z, Choudhry AM, Ikram H, Asghar MW, Kajal D, Waheed A, Mobassarah NJ. Ann. Neurosci. 2013; 20(4): 157-161.

Copyright

(Copyright © 2013, Indian Academy of Neurosciences)

DOI

10.5214/ans.0972.7531.200409

PMID

unavailable

Abstract

The development of eating disorders including anorexia nervosa, bulimia nervosa, binge eating disorder, and atypical eating disorders that affect many young women and even men in the productive period of their lives is complex and varied. While numbers of presumed risk factors contributing to the development of eating disorders are increasing, previous evidence for biological, psychological, developmental, and sociocultural effects on the development of eating disorders have not been conclusive. Despite the fact that a huge body of research has carefully examined the possible risk factors associated with the eating disorders, they have failed not only to uncover the exact etiology of eating disorders, but also to understand the interaction between different causes of eating disorders. This failure may be due complexities of eating disorders, limitations of the studies or combination of two factors. In this review, some risk factors including biological, psychological, developmental, and sociocultural are discussed.

Language: en

Keywords

Canada; human; suicide; incidence; survival; immigration; prevalence; lifestyle; sex difference; cause of death; mortality; suicide attempt; personality; hospitalization; anorexia nervosa; binge eating disorder; body image; risk assessment; comorbidity; disease severity; serotonin 1A receptor; Anorexia nervosa; Bulimia nervosa; risk factor; serotonin; article; bulimia; eating disorder; psychological aspect; disease association; rating scale; pathophysiology; fear; physical examination; cultural factor; disease course; age distribution; Etiology; anxiety disorder; environmental factor; 5 hydroxyindoleacetic acid; dopamine; neurotransmission; electrolyte disturbance; starvation; economic aspect; social status; child sexual abuse; weight gain; self esteem; irritability; cardiovascular disease; anabolic agent; satisfaction; underweight; anemia; hematologic disease; gastrointestinal disease; body weight; endocrine disease; obsessive compulsive disorder; convalescence; serotonin transporter; twins; psychosocial withdrawal; physical abuse; cost of illness; body mass; heredity; protein function; serotonin 2A receptor; child neglect; amenorrhea; Co-morbidity; cachexia; mesolimbic dopaminergic system; Binge eating disorder; protein cerebrospinal fluid level; nutritional intolerance; eating habit; atypical eating disorder; Eating Disorder Inventory