Citation

San Lorenzo IS, Chiesa M, Gamba P, Toniolo A. Cardiologia 1989; 34(5): 439-446.

Vernacular Title

Aspetti cardiologici dell'intossicazione da monossido di carbonio.

Copyright

(Copyright © 1989, Società Italiana di Cardiologia)

DOI

unavailable

PMID

2758446

Abstract

Carbon monoxide poisoning causes tissue hypoxia because of reduced transfer and altered release of oxygen by hemoglobin. Considering many case histories, we realized that symptoms and clinical signs of acute poisoning are mostly neurologic: coma, headache, dizziness, vomiting. On the contrary, it seems that myocardium, the other organ which mostly requires O2, is attacked in a "silent way". ECG in 5 patients with accidental carbon monoxide poisoning underlined that cardiac rate increased (3 of them presented tachyarrhythmias by atrial fibrillation) and the presence of more or less important alteration of ventricular repolarization like "subendocardial lesion". Simple hyperbaric oxygen treatment determined the regression of the rhythm disorder and of the abnormalities of ventricular repolarization. The only patient who had not the restoration of sinus rhythm had chronic atrial fibrillation.

Language: it